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Sinus Problems

Dr Jason Roth (MED0001185485) — Specialist Otolaryngologist & Head and Neck Surgeon, specialist registration in Otorhinolaryngology, Head & Neck Surgery.

The paranasal sinuses — four pairs of air-filled cavities within the skull bones surrounding the nose and eyes — are among the most common sources of symptoms managed in ENT practice. Problems range from a single acute infection following a cold to severe, chronic, treatment-resistant disease significantly affecting quality of life. This page provides a comprehensive overview of the range of sinus conditions, their mechanisms, and the treatment approaches available. Detailed information on specific conditions is available through the links below.


Anatomy — The Paranasal Sinuses

The four pairs of paranasal sinuses are:

  • Maxillary sinuses: In the cheekbones. The largest sinuses and the most commonly involved in acute sinusitis. Drain through a high posteromedial opening that makes gravity-dependent drainage difficult.
  • Ethmoid sinuses: A labyrinth of small air cells between the eyes and nose. Critically important in sinus disease — the anterior ethmoids are the “key” to sinus drainage, and ethmoid disease is present in virtually all patients with chronic rhinosinusitis.
  • Frontal sinuses: Above the eyes in the frontal bone. Drain through the narrow frontal recess — the most difficult drainage pathway to access and maintain in sinus surgery.
  • Sphenoid sinuses: Deep in the skull base, adjacent to the pituitary gland, optic nerves, and carotid arteries. Isolated sphenoid disease is uncommon but can produce deep, poorly localised headache.

All sinuses drain into the nasal cavity through small openings. The ostiomeatal complex (OMC) — a convergence of the drainage pathways of the maxillary, anterior ethmoid, and frontal sinuses in the lateral nasal wall — is the anatomical keystone of sinus disease: obstruction here produces downstream failure of three pairs of sinuses simultaneously. The OMC is the primary target of endoscopic sinus surgery.


How Sinus Problems Develop

Normal sinus health requires two elements: patent drainage pathways and effective mucociliary clearance — the coordinated beating of microscopic cilia on the sinus lining that continuously sweeps the mucus blanket toward the ostia and into the nasal cavity. When either element fails, the consequences are predictable:

  1. Mucus accumulates in the sinus, providing a nutrient-rich culture medium for bacteria
  2. Reduced oxygen tension in the obstructed sinus favours anaerobic bacterial growth
  3. The inflammatory response to infection further swells the mucosal lining, worsening obstruction
  4. Repeated cycles of obstruction and infection progressively damage the mucosal lining and impair healing

The triggers for this cascade include viral upper respiratory infections (the most common precipitant of acute sinusitis), allergen exposure causing mucosal oedema and turbinate swelling at the OMC, structural anatomical variants (concha bullosa, Haller cells, deviated septum) that narrow the drainage pathways, and underlying systemic conditions (aspirin sensitivity, primary ciliary dyskinesia, immunodeficiency, cystic fibrosis) that impair mucociliary or immune function.


Range of Sinus Conditions

Acute rhinosinusitis
Sinus inflammation lasting fewer than 12 weeks, most commonly following viral URTI. Up to 90% of acute episodes are viral and self-resolving. Bacterial superinfection is suggested by symptoms persisting beyond 10 days, severe symptoms, or worsening after initial improvement. Read more about sinusitis →

Chronic rhinosinusitis (CRS)
Sinonasal inflammation persisting for 12 weeks or more, affecting an estimated 10–12% of adults. Divided into CRS without nasal polyps (neutrophilic, often structural) and CRS with nasal polyps (eosinophilic, type 2 inflammatory). Both require objective confirmation by endoscopy or CT. Read more about chronic rhinosinusitis →

Nasal polyps
Benign inflammatory growths arising from the sinus lining. Associated with eosinophilic inflammation, allergic rhinitis, aspirin sensitivity, and asthma. Cause progressive nasal obstruction, anosmia, and recurrent infection. Highly steroid-responsive but often recur. The new biological agents (dupilumab) represent a significant advance for severe polyp disease. Read more about nasal polyps →

Sinus headaches
Facial pain from sinus obstruction or infection. Importantly, the majority of self-diagnosed “sinus headaches” are actually migraine — a distinction with major implications for treatment. True sinus headache requires objective evidence of sinus disease. Read more about sinus headaches →

Allergic rhinitis and its contribution to sinus disease
Allergic rhinitis — affecting 18% of Australians — drives turbinate hypertrophy, nasal oedema at the OMC, and impaired mucociliary clearance. It is the most common underlying condition in patients presenting with chronic sinus disease, and optimal allergy management is an essential component of CRS treatment. Read more about allergy treatment →

Post-nasal drip
The sensation of mucus flowing from the sinuses and nose into the back of the throat — causing throat clearing, chronic cough, and throat irritation. Often the dominant symptom of CRS, allergic rhinitis, or non-allergic rhinitis. May also relate to LPR (laryngopharyngeal reflux) or medications. Read more about post-nasal drip →


Symptoms of Sinus Disease

The cardinal symptoms of rhinosinusitis, as defined by EPOS, are:

  1. Nasal obstruction or blockage — from mucosal oedema, turbinate hypertrophy, polyps, or anatomical obstruction
  2. Nasal discharge or post-nasal drip — anterior discharge (running from the nose) or posterior (draining into the throat)
  3. Facial pain or pressure — location reflecting the affected sinus; maxillary (cheeks), ethmoid (between eyes), frontal (forehead), sphenoid (deep/vertex)
  4. Reduction or loss of smell (hyposmia/anosmia) — particularly associated with nasal polyps

Additional symptoms frequently reported include headache, chronic cough, fatigue, post-nasal drip, ear fullness (from Eustachian tube effects), and altered taste. The SNOT-22 (Sinonasal Outcome Test) is a validated questionnaire used to quantify sinonasal symptom burden and monitor treatment response.


Investigation

Nasal Endoscopy

The cornerstone of ENT assessment for sinus disease. A rigid telescope passed through each nostril allows direct visualisation of the nasal septum, turbinates, middle meatus, olfactory cleft, and nasopharynx. The presence of nasal polyps, purulent middle meatal discharge, mucosal oedema at the OMC, or anatomical obstruction can all be identified and directly informs the diagnosis and management plan. Endoscopy is essential and significantly superior to simple anterior nasal examination with a speculum.

CT Sinuses

High-resolution CT of the paranasal sinuses provides the anatomical road map for surgical planning. It documents the extent and distribution of mucosal disease, the anatomy of the ostiomeatal complex and frontal recess, the degree of septal deviation, the presence and extent of anatomical variants, and any complications. CT should be performed at a stable baseline rather than during an acute infection, which causes non-specific mucosal thickening. CT is mandatory before any sinus surgery.

Allergy Testing

Skin prick testing or specific serum IgE measurement identifies atopic sensitisation contributing to chronic sinonasal disease. Documentation of specific allergens allows targeted avoidance advice and guides immunotherapy planning.

Olfactory Testing

Objective smell testing (UPSIT 40-item or Sniffin’ Sticks) documents the degree and type of olfactory loss, distinguishing conductive (obstructive) from sensorineural loss. Baseline documentation is important before any surgical or medical intervention that may affect olfaction.


Treatment Approaches

Medical Treatment

  • Intranasal corticosteroid sprays (first-line for all CRS phenotypes)
  • High-volume saline nasal irrigation (daily, grade A evidence)
  • Antibiotics — for acute bacterial sinusitis; prolonged low-dose macrolides for CRSsNP
  • Short-course oral corticosteroids — for CRSwNP and severe acute episodes
  • Antihistamines — for allergic rhinitis component
  • Allergen immunotherapy — for documented allergic rhinosinusitis
  • Biological agents (dupilumab, mepolizumab) — for severe uncontrolled CRSwNP
Surgical Treatment

  • FESS — for CRS and polyps not controlled by medical treatment
  • Septoplasty — for deviated septum obstructing drainage or breathing
  • Turbinoplasty — for inferior turbinate hypertrophy
  • Nasal polypectomy — for localised or extensive polyp removal
  • Modified Lothrop (Draf III) — for complex frontal sinus disease

For detailed information on specific conditions:

Contact us to arrange a consultation →

Dr Roth’s Clinical Perspective

The sinuses are a source of genuine misattribution in medicine — many symptoms that patients and GPs attribute to sinus disease have other causes, and the investigation should be directed at confirming the diagnosis rather than assuming it from the symptom description. Headache alone is rarely sinusitis. Post-nasal drip alone may be reflux. Nasal obstruction alone may be a deviated septum or turbinate hypertrophy. The combination of two or more cardinal sinus symptoms with objective evidence on CT or endoscopy is what confirms the diagnosis and justifies treatment. Getting to that point before committing to surgery is the most important part of sinus management.

— Dr Jason Roth, MBBS, FRACS (ORL-HNS), IBCFPRS

Dr Jason Roth — Specialist Otolaryngologist Sydney

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Dr Roth consults from Dee Why on Sydney’s Northern Beaches. A GP referral is recommended.

Dr Jason Roth (MED0001185485) — Specialist Otolaryngologist & Head and Neck Surgeon.

Dr Jason Roth | MBBS, FRACS (ORL-HNS) | MED0001185485
Specialist Otolaryngologist & Head and Neck Surgeon
Specialist registration — Otorhinolaryngology, Head & Neck Surgery
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